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Engages GLP-1 receptors in the hypothalamus and hindbrain to reduce hunger, increase satiety, and lower overall energy intake the primary driver of the weight reduction observed in the STEP trial program

The research is ongoing and largely based on non-human studies

T3 suppression on GLP-1 / GIP / glucagon agonism caloric deficit and hepatic energy expenditure together downregulate T4-to-T3 conversion

Mol Neurodegener 12(1):64

When B12 is paired with lipotropic compounds, the support extends to liver pathways that determine how efficiently fat is broken down, transported, and converted

In this blend they are already combined, and the pairing has a mechanistic rationale: NAD+ supplies the coenzyme, while 5-Amino-1MQ blocks the NNMT enzyme that would otherwise drain NAD+ precursors [4]