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Thats because these approaches often only suppress inflammation rather than address the real issue: cellular degeneration in the tendons

for MetS and T2D), and because exercise is one of the most potent IGF-1 synthesis/freeing mechanisms, it seems logical to correlate them and establish IGF-1 as a target for future options in the multifactorial treatment for metabolic syndrome

doi: 10.1016/j.biopha.2015.07.025, PMID: [DOI] [PubMed] [Google Scholar] 43.Borkum JM

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