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Although higher oxidative stress and an upregulation of amyloid (A) peptides from an extra copy of the APP gene are attributed to the AD susceptibility, the relationship between the two factors is unclear

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As GLP-1 has been implicated as a mediator in the putative gutrenal axis (a rapid-acting feed-forward loop that regulates postprandial fluid and electrolyte homeostasis), direct actions on the kidney have been proposed

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