Gong et al., 2016) have underscored the potential of TMP as an innovative therapeutic option in CI-AKI prevention by suppressing the p38 MAPK and FoxO1 pathways, additionally protecting renal tubular cells against arsenite-induced nephrotoxicity by averting mitochondrial dysfunction and regulating autophagy
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Molecular basis for the distinct functions of redox-active and FeS-transfering glutaredoxins
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