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The AKT/mTOR hyperactivation in these organoids suggests that mTOR inhibitors (e.g., rapamycin analogs) may ameliorate synaptic imbalances, warranting clinical exploration in ASD subtypes with CNTNAP2 mutations

B., & Seghinsara, A

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Disruption of leukemia/stroma cell interactions by CXCR4 antagonist CTCE-9908 enhances chemotherapy-induced apoptosis in AML

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