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These results highlight the importance of further exploring the mechanisms underlying organ-specific aging, informed by established hallmarks of aging, which could pave the way for effective interventions aimed at enhancing healthspan and mitigating negative health outcomes related to aging

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This interaction helps cells move quickly, reshape their internal framework, and repair damaged tissues

[5] Disruption of these pathways leads to accumulation of homocysteine and methylmalonic acid, metabolic signatures linked to megaloblastic anemia and demyelinating neuropathy

It normalized markers such as malondialdehyde, nitric oxide, IL-6, and specific matrix metalloproteinases while increasing superoxide dismutase and IL-10 in the affected nerves [103]