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Using genome-wide chromosomal analysis methods, Celestino-Soper and coauthors[43] identified 55 potentially pathogenic copy number variants, among those and in the male samples, a deletion of exon 2 of the TMLHE gene (trimethyllysinehydroxylase epsilon) was found, this gene encodes the first enzyme in the biosynthesis of carnitine which is located in mitochondria

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The precursor mass tolerance was set to 20 ppm, while the fragment mass tolerance was set to 0.02 Da

The results from the PBMC proliferation assay demonstrated a reduced proliferation on 42 d pp compared to 42 d ap , indicating a different sensibility of PBMC

10.1016/j.jnutbio.2006.12.003 [DOI] [PubMed] [Google Scholar] 51.Vali S, Mythri RB, Jagatha B, Padiadpu J, Ramanujan KS, Andersen JK, et al

Advances in Gerontology, 2015