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It is the most common cause of endogenous hyperinsulinemic hypoglycemia in adult patients without diabetes and might play a part in the multiple endocrine neoplasia type 1 (MEN1) syndrome 2

Douros, A., et al

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Importantly, IGF-I and its binding proteins exert acute anabolic effects on metabolism as well as mediating myoblast proliferation, differentiation, and apoptosis[2]