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Moreover, little is currently known about the downstream effectors of eryptosis-associated kinases

We observed significantly enhanced tumor control reflected by longitudinal bioluminescence imaging (Fig

Activated mutations within this pathway represent the most frequent oncogenic factor for many cancers (Khotskaya et al

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While density for the N-terminal lipidation was not visible in the lower resolution cryo-EM map of the repeat Cagri-CTR-Gs complex ( N = 2), the outward rotamer of H296 ECL2 was observed, congruent with similar interactions between the N-terminal acylation and ECL2