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Estrogen has also been found to reverse chemotherapy resistance of ovarian cancer by targeting the Nrf2/GPX4 signaling axis to induce ferroptosis in drug-resistant human epithelial ovarian cancer cells (74)

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These two distinct phenotypes also perform different functions: the pro-inflammatory classically activated M1 phenotype exerts neurotoxic effects, while the anti-inflammatory alternatively activated M2 phenotype plays a neuroprotective role (37)

Moreover, oxidative stress modifies EVs cargo, enabling them to carry oxidants, antioxidants, or redox-related enzymes, thus influencing redox homeostasis in target cells either positively or negatively [171]

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