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IGF-1 LR3 is a synthetic peptide analog structurally derived from insulin-like growth factor sequences, modified through amino acid substitution and extension to alter receptor interaction dynamics and resistance to enzymatic degradation

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Reduced SAM levels due to methionine starvation promote the dissociation of SAM with SAMTOR and then promote the association of SAMTOR with GATOR1 to repress the mTORC1 pathway