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The molecule itself is fragile compared to small-molecule medications

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[3] The mechanism driving muscle loss during prolonged caloric restriction involves increased muscle proteolysis rather than suppressed protein synthesis, [4] meaning the body actively breaks down existing muscle tissue to meet energy demands, and this process accelerates in hormone-deficient states

While cagrilintide is not yet FDA-approved , its ongoing development suggests strong potential as part of a new pharmacologic framework in obesity care