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By incorporating checkpoint inhibitors (e.g., anti-PD-1/PD-L1 antibodies), cytokines, or innate immune activators such as STING agonists into hydrogel scaffolds, the suppressive cervical TME can be actively reprogrammed into a pro-inflammatory and immunostimulatory state [62,63,64]

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Concurrently, emerging computational tools such as METALICA (Ruiz-Perez et al., 2024) could be applied to infer microbial gene regulatory networks and quantify trait heritability from metagenomic data, thereby elucidating the interplay between host genetics and environmental exposure

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Shaman AM, Bain SC, Bakris GL, Buse JB, Idorn T, Mahaffey KW, et al