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The older we get, the less growth hormone we produce and the more our neurons degenerate

Among these, NRF2 has been shown to play a critical role in regulating intracellular proteolytic degradation systems (i.e., the proteasome and autophagy-lysosome pathway) as well as molecular chaperones designed to maintain proper protein conformation and stability

Small-molecule compounds, termed senolytics, which increase the death rate of senescent cells, can alleviate inflammation and age-related organ dysfunction by targeting diverse survival pathways

It is plausible to postulate that the first and second GSH molecule should interact during the reductive half-reaction of the dithiol mechanism with the 1st and 2nd glutathione-interaction site, respectively

Garofalo V et al

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