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c , Methylarginine and NDUFS2 western blotting of immunoprecipitated NDUFS2 in Control, Bmal1 / and Cry1/2 / MEFs (n = 2)

The BSO treatment of HepG2 cells that causes marginal GSH deficiency increases ceruloplasmin synthesis due to increased transcription mediated by activator protein (AP)-1-binding site [110]

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Note also that GSH inhibits both GCL and GS, so it is not clear by inspection of the diagram what the effect of raising the activities of GCL and GPX will be