Statements Data availability statement The data that support the findings of this study are openly available in ZENODO: Supplementary Data 1: Supplementary Data 2: Author contributions YH-O: Conceptualization, Data curation, Formal Analysis, Funding acquisition, Investigation, Methodology, Project administration, Resources, Software, Supervision, Validation, Visualization, Writingoriginal draft, Writingreview and editing
This special binding could be explained by the colocalization of mGluR1 and A and the presence of A and GluN2B immunoreactivities in neurites [35, 36] In AD, AO prevents physiological activation of the cellular prion protein-mGluR5 (PrPC-mGluR5) complex by glutamate
Researched alongside the GH/IGF-1 axis, which is associated with muscle and body-composition biology
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Ohguro H, Oghuro I, Katai M, Tanaka S
Furthermore, studies have reported an aberrant activation of NRF2, a key antioxidant transcription factor involved in the regulation of critical antioxidants including glutathione, SOD, and CAT, leading to reduced antioxidant capacity (Davinelli et al., 2025) In ASD children, the oxidative stress is not only higher, but it manifests in a complicated new pattern concerning different types of ROS and antioxidants (Hirayama et al., 2020)