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This is where a therapy like MOTS-c becomes relevant to the conversation

Eligibility comes first, every time

The doctor can perform blood tests to check which deficiency they might have, and recommend the best treatments

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Because GLP-2 acts through subepithelial myofibroblasts, which are the same cell population implicated in intestinal fibrogenesis, the risk-benefit profile of GLP-2 agonism may differ substantially between mucosal UC, inflammatory ileal CD, and stricturing or penetrating CD phenotypes, and this phenotypic stratification should be a required feature of future trial design

doi:10.1371/journal.pgen.1002325