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Similar to dNK cells, miR-483-3p inhibitor-transfected NK cells derived from UCB/CD34 + HSCs demonstrated upregulation of perforin and CD107a (Supplementary Fig

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In this study, we demonstrated that the correction of glycine deficiency with dietary supplements increased the urinary excretion of acylglycine derived from the intermediates of the BCAAs (isobutyrylglycine, tigylglycine, and isovalerylglycine) and fatty acid oxidation (hexanoylglycine)