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Considering that P1 in rats corresponds to approximately 23 weeks gestation in humans, and P8 around delivery (101), these findings suggest that mid-gestational exposure to VPA produces excitatory-dominant E/I imbalance by blocking the excitatory-to-inhibitory shift of GABAergic signaling or reducing VGAT expression and decreasing GABA synthesis in the postnatal brain

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This is the opposite of the antagonistic relationship seen with ibuprofen

The most common disorder is X-linked sideroblastic anemia (XLSA), caused by mutations in 5-aminolevulinic acid synthetase 2 (ALAS2), the initial rate-limiting step in erythroid heme synthesis

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