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In both control and BPM31510-treated MIA PaCa-2 and PANC1 cells, pyruvate (Complex I)-specific H 2 O 2 production was minimal, indicating that Complex I is an unlikely site for the exacerbated ROS production observed after BPM31510 treatment (Fig

Weivoda, M

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It makes sense for both researchers and supplement companies to focus on NMN and NR, as the salvage pathway is responsible for so much NAD+ production

When protein synthesis was blocked by CHX, sparing of cysteine from protein translation was sufficient to reactivate mTORC1 and relieve the ISR (Fig