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Smoking, nicotine, and ACE2 On one hand, it is proposed that smoking or nicotine may upregulate the detrimental angiotensinconverting enzyme (ACE) but downregulate the compensatory ACE2/ANG(17) receptor axis and hence contribute to cardiovascular and pulmonary diseases [53, 54, 55]

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I have been fortunate to be involved in several early studies of treatment of people seeking help with tobacco dependence and this provided a starting point for a body of research that is still on-going

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