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Collectively, these results indicate that the Tg(h GPR15 ) phenotype with elevated GPR15 levels could aggravate TNBS-induced colitis while offering better protection against DSS-induced ulcerative colitis under cigarette smoke exposure

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As the present study might lack sufficient power to characterize predictive instruments for smoking continuation (see Table 1), future studies leveraging even larger sample sizes for both smoking continuation and PD will be needed to validate these findings

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