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The genetic basis of the reduced expression of bilirubin UDP-glucuronosyltransferase 1 in Gilberts syndrome

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HMs induce excess reactive oxygen (ROS) species that lead to oxidative stress-induced cellular toxicity, and lipid peroxidation in plant cells [7]

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Complement proteins C1q and C3 are increased in MS, and can be seen at synapses located within HLA-positive cell processes and in lysosomes, indicating opsonization and engulfment by microglia (150)