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In addition, TJ proteins were also significantly suppressed in lung tissue of patients with end-stage COPD and in airliquid interface differentiated epithelial cells from these patients [13]

3.1 Endothelial dysfunction Various clinical and laboratory studies have shown that endothelial dysfunction induced by cigarette smoke is mediated by decreased nitric oxide (NO) bioavailability, increased production of superoxide anions, and increased production and release of endothelin ( 28 )

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