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This hypothesis might have been corroborated by the observation that the levels of the cleaved forms of caspase-3 and PARP, both proteins which may be associated with programmed cell death, seemed to be reduced after introduction of AHK-Cu

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However, excess APAP intake results in the rapid depletion of cellular GSH stores, followed by hepatic damage as hepatocytes undergo cell death 23,24,25

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