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Studies show that 50% of young men and 10% of young women start smoking very early,5,6 and three out of five youths who smoke cigarettes will grow up to become adult smokers.7,8 Adolescence is recognizably a key period of social vulnerability to the adoption of risk behaviors.7 Even though teenagers are aware of these risk situations, they end up adopting behaviors and attitudes likely to compromise their health,9,10 and most habits acquired during this life stage are carried on well into adulthood, being extremely hard to break.11 During the past years, different theories and models had been proposed to explain the influence that social context and environment have on youth behavior and tobacco use initiation,12 such as individual factors (age, low self-esteem, perception of risk),13,14 family factors (parental smoking, educational level, parental attitude toward tobacco),15,16 social factors (peer groups),17 school environment (debates, smoking prevention programs, lack of school-based anti-tobacco policies),18,19 environmental factors (advertising, availability),20 and tobacco industry promotional and advertising activities.21,22 Moreover, adolescent smokers consume three times more alcohol, use eight times more marijuana, and 22 times more cocaine than their non-smoking peers

Heme oxygenase-1 in human brain tissue To determine whether HO-1 levels are altered in human PD brain tissue, we evaluated HO-1 in anterior cingulate cortex samples from PD patients harboring neuropathologically proven Lewy body disease, Alzheimers disease (AD), or age-matched normal controls