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The irreversible transformation of adenosine to hypoxanthine is another reason for the low efficiency of ATP regeneration in GSH biosynthesis [26]
These pathways are, then, able to induce an abnormal increase in free intracellular calcium in their targets by activating non-NMDA glutamate gated ion channels as well as G protein-coupled receptors, which are capable of mobilizing calcium from internal stores (Sharp et al
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