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GH then travels to the liver, activating GHR-Jak2-STAT5b signalling, upregulating IGF-1 synthesis (predominantly IGF-1Ea/1Eb splice variants), and inducing hepatic IGFBP-3 and ALS (acid-labile subunit) to form the circulating 150kDa ternary complex that extends IGF-1 half-life to 12-15 hours in the systemic circulation

Nutritional interventions for mitochondrial OXPHOS deficiencies: mechanisms and model systems

Kinetic mechanism for viral dsRNA length discrimination by MDA5 filaments

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Behrendt, D

Cellular senescence and the senescent secretory phenotype: therapeutic opportunities