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Despite reports of cell Golgi stress and dysregulation of Golgi-associated genes in T1D, as well as the role of the Golgi in class I HLA hypo-expression following CVB infection, the effects of CVB infection on islet Golgi integrity are poorly studied (246)

As an example, GSH depletion has been shown to promote pro-inflammatory microglial phenotypes and amplify astrocyte-microglia crosstalk ( Given these roles, GSH has emerged as a promising therapy and biomarker in neurodegenerative diseases

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