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Itraq-based quantitative analysis of hippocampal postsynaptic density-associated proteins in a rat chronic mild stress model of depression
This is where I think the best path forward is to present several theories, which Im listing roughly from plausible to speculative
Degradation of glucagon-like peptide-1 by human plasma in vitro yields an n-terminally truncated peptide that is a major endogenous metabolite in vivo
These findings provide new perspectives on RA pathology and identify potential therapeutic targets for addressing inflammatory and immune imbalances in RA
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