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SL significantly inhibited the intrahepatic synthesis of several disease-related pro-inflammatory cytokine mRNAs, i.e., for TNF, interferon-gamma (IFN-g), IL-4, and IL-2, intrahepatic induction of iNOS, and intrahepatic activation of NF-B (Morishima et al., 2010

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GSH is thought to be the most important endogenous non-enzymatic antioxidant (Kurutas 2016) and hence may be a key target in future treatments for MDD

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