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While a fibrotic matrix initially contributes to tissue repair, after injury, chronic and persistent kidney damage in the development of CKD leads to uncontrolled fibrotic matrix deposition, resulting in reduced blood supply, destruction of normal organ structure, and eventual glomerulosclerosis, renal tubular atrophy, and interstitial fibrosis [9, 10]
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Jentzsch V, Osipenko L, Scannell JW, Hickman JA
European Journal of Applied Physiology, 73, 434-439
Whether this is due to the limited increase in GLP-1 from 3.3 pM 0.5 pM (before LFD) to 3.6 pM 0.9 pM (after LFD) should be investigated in future studies
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