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Identifying these can reveal novel therapeutic targets for epilepsy management

25HC-integrin-FAK amplifies proinflammatory response To examine whether extracellular 25HC can trigger proinflammatory response in mice, we administered (via intraperitoneal route) 25HC to C25H KO mice, as they do not produce any 25HC and the observed response will be entirely due to exogenously added 25HC

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Historically, the use of beta-blockers in individuals with ATTR-CM raised concern because of dependence on heart rate to maintain cardiac output, chronotropic incompetence from amyloid infiltration of the conduction system, and autonomic dysfunction